What causes type-1 diabetics to stop producing insulin? The mainstream theory sounds lacking to me. As the U.S. Library of Medicine writes: “an infection or another trigger causes the body to mistakenly attack the cells in the pancreas that make insulin.”
I’d interpret that to mean that a virus or bacteria invaded the body. The cellular structure of the invader was similar to the islet cells of the pancreas. So the immune system attacks both the invader and the pancreas. When the invader leaves, the immune system doesn’t notice. It continues to attack the islet cells for the rest of the type-1’s tortured life.
It’s a sound theory. But just a theory.
I recently discovered Dr. Lawrence Wilson’s article Diabetes, a 21st Century Epidemic. Here he offers a slightly different explanation. “[Type-1 diabetes] is usually due to an infection of the pancreas that stops or reduces insulin production.”
Dr. Wilson’s explanation sounds similar to the mainstream explanation, but suggests that the infection remains in the pancreas. This would explain why the immune system wants to hammer it day after day.
“This infection cannot be detected on standard blood tests, but the rapid onset often leads one to such a conclusion,” writes Dr. Wilson. I guess if the immune system is doing such a good job at isolating the infection within the confines of the pancreas, the infection might appear in the blood.
While such an infection doesn’t appear on blood tests, Dr. Wilson says that “hair analysis may show an infection pattern, though often it does not at first.” This refers to a special method developed by the late Dr. Paul Eck to diagnosis deep-rooted health conditions.
The theory is more supported by his clinical experience. When patients are treated for an infected pancreas, insulin production returns. At least that’s how I interpret his article.
Could not researchers dissect the pancreas of deceased type-1 diabetics? In this way, I’d assume, they could determine whether an infection exists or not.
“Usually, this type of diabetes, ” continues Dr. Wilson, “starts after a mild cold or flu, or some other innocuous infection, and it can develop suddenly.” My wife, Nicole, developed diabetes after a bout with the mumps. Another friend, developed diabetes after suffering the worst diarrhea of his life in East India.
The idea that an infection lives on in the pancreas seems sound to me. Far better than the idea that the immune system has gone suicidal. Of course, there may be multiple causes of type-1 diabetes, including Dr. Campbell-McBride’s theory that it’s related to leaky gut syndrome.
“The infection,” continues Dr. Wilson, “may be triggered or related in some way to excessive iron, manganese or aluminum in the pancreas.”
Iron bacteria, according to Wikipedia, can thrive off iron or manganese. This is a well known problem amongst water management in cities that rely on well water. It stands to reason that if the pancreas became overloaded with iron or manganese, that iron-feeding bacteria would set up camp. The immune system, trying to protect the pancreas, launches an attack. In the biological warfare the follows, the islet cells of the pancreas are destroyed. War is always rough on infrasrtucture.
Or maybe, the immune system has nothing to do with the destruction of the iselt cells. Maybe it’s all the fault of infection. The immune system is simply trying to get rid of the deeply hidden infection. Because the infection is so well hidden, the defending immune system gets blamed for the destruction of the islet cells instead.
In future posts I’ll write about how this concept of infected and toxic pancreas has dramatically altered the experiment we are conducting on Nicole to cure her type-1 diabetes.
You can read more about Dr. Wilson’s views and treatments for type-1 diabetes in his article: Diabetes, a 21st Century Epidemic.